Agensis of corpus callosum
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Agenesis of the Corpus Callosum
Kara Rood
100043820
Acadia University
Psyc 3833X1
November 5, 2003
Throughout history many infants have been asymptomatic for certain brain malformations such as Agenesis of the Corpus Callosum (ACC). It has not been until the children became of school age before the onset of different symptoms such as intellectual impairments, drowsiness, difficulties at school and seizures were apparent. However, with advancements in neurological diagnosing techniques, such as fetal screening and MRI, the prognosis of such neurological diseases can now to be detected prenatally (Jamroz, Kluczemska, & Marszak, 2000).
Agenesis of the Corpus Callosum was first described in an autopsy in 1812. It is a
neurological disorder that results from the absence or partial absence of the corpus
callosum, the most important commissural structure in the brain. This structure consists
of a cluster of transverse fibers that connect the right and left hemispheres of the brain.
The corpus callosum is located just above the thalamus and below the cerebrum and is
divided into the rostrum, genu, body and splenium ( Pinel, 2000).
The corpus callosum begins to develop between the 10th and 25th week of gestation. It
acts as a bridge to transfer in interhemispheric information by connecting matching
neocortical areas that allow fibers to carry messages for integrated motor, sensory, and
cognitive performance between left and right hemispheres. Despite the apparent
importance of the corpus callosum, evidence has shown that it is not needed for life or
good health in some cases ( Adamshaum, Andre, Bogaert, Feingold, Gelot, Kieffer et al,
2003)...